Are COVID Deaths Boosted by Micro-Dose Nerve Gas, Micro-Drone Warfare?

VT: Low dosage Sarin gas was tested on the New York subway system more than a decade ago.  People got sick.  We believe that the test and others along with studies listed below and information from Senate hearings provide sufficient backdrop to support what everyone is coming to see.

Data on tests inside the US has crept to the surface from time to time, but, during the Cold War, every imaginable disease, human or plant, every fungus, rust, virus or bacteria that could cripple the economies of nations like China, Russia, Cuba, Syria, Venezuela, Nicaragua, Vietnam..

Oh, how about Agent Orange and 1 million dead Americans, even more than that murdered with COVID…and millions more Vietnamese who are still dying to this day.

Some of that information that doesn’t exist and off course none of the things we deserve to see on a war waged against the world, using biological and chemical warfare not just for globalism and politics but out of worship of cruelty itself.

From SOTT 2016:

Monday morning, scientists working for the Department of Homeland Security will begin releasing (nontoxic) gases and particles on crowded subway platforms, beginning a week-long airflow study aimed at measuring the impact of a nightmarish chemical or biological terrorist attack.

It’s not the first such study — most recently the NYPD gassed the subway system in 2013 (also nontoxic) — but this is the first large-scale use of particles, in addition to gases. While gas tests help scientists, counter-terrorism specialists, and emergency responders understand the impact of chemical weapons like sarin gas or mustard gas, the particle test will measure the fallout from aerosol-dispensed biological agents like anthrax or ricin.

Every day this week, particles will be released from machines at busy stations like Grand Central, Times Square, 34th St. – Penn Station. Special machines and filters on platforms and subway cars will gather the particles. Researchers working throughout the system will also wear small patches designed to collect them.

The Department of Homeland Security, along with a MTA and a slew of other agencies, assured New Yorkers that neither the gases nor the particles are a health risk. The study won’t significantly increase the level of particle matter wafting through the air, in part, because New York City subway stations are already brimming with particulate. In fact, as the project’s assessment report noted, the levels of steel, manganese, and chromium in the subway system are 100 times higher than outdoors.

This report is from the Murdoch/Fox owned New York Post:

A declassified FBI report from the Baltimore field office dated Aug. 25, 1950 provides some tantalizing support for the claim. “The BW [biological weapon] experiments to be conducted by representatives of the Department of the Army in the New York Subway System in September 1950, have been indefinitely postponed,” states the memo, a copy of which the author provided to The Post

An Olson colleague, Dr. Henry Eigelsbach, confirmed to Albarelli that the LSD subway test did, in fact, occur in November 1950, albeit on a smaller scale than first planned. Little, however, is known about the test — what line, how many people and what happened.

The purported experiment occurred nearly a year before a more infamous August 1951 incident in the small town of Pont St. Esprit, in the south of France, when the citizens were hit by a case of mass insanity.

Over a two-day period, some 250 residents sought hospital care after hallucinating for no apparent reason. Thirty-two patients were hauled off to mental asylums. Four died. Mercury poisoning or ergot, a fungus of rye bread, was cited as the culprit. But ergot is also one of the central ingredient of LSD. And curiously enough, Olson and his government pals were in France when the craziness erupted.

This is from Business Insider, 2015:

On June 6, 1966, a group of US Army scientists made their way into the Seventh and Eighth Avenue lines of the New York City subway. Some carried air sampling machines in boxes and on belts; others carried light bulbs.

The light bulbs were packed with about 175 grams of Bacillus subtilis bacteria, then known as Bacillus globigii — approximately 87 trillion organisms in each. The plan was to shatter them and then use the sampling machines to see how they spread through the subway tunnels and trains.

This test was one of at least 239 experiments conducted by the military in a 20-year “germ warfare testing program” that went on from 1949 to 1969. These experiments that used bacteria to simulate biological weapons were conducted on civilians without their knowledge or consent. That stands in direct violation of the Nuremberg Code, which stipulates that “voluntary, informed consent” is required for research participants.

Is All COVID real COVID?

Nobody is dying in Vietnam.  Dozens of other nations whose population resembles the US in many ways are having no pandemic.  Does this suggest targeting or does it suggest something more sinister, that the inexplicable array of wildly divergent COVID symptoms may be “wall paper” disguising something else?

This story is from August 2020:

It isn’t just Italy, New York is an epicenter too and it isn’t simply CV 19 but chemical weapons that mimic the disease, according to highest level intelligence sources.

Defense experts are seeing a targeted biowar on China, Italy, Spain, Iran and left-leaning political regions of the US.  We will now discuss our findings.

In advanced nations, the overall death rate compared to those identified as infected averages under 1%.  In Italy and Iran, the rate is many times that and the reasons are given are fake.  Both nations have very advanced medical capabilities.

Then we look at Spain, China’s best friend in Europe and, of course, China.

Moreover, reporting on CV19 includes concocted conspiracies that trace to Israeli intelligence and the CIA, spread by Trump and Pompeo, which are impossibly absurd and hide a multifaceted operation thoroughly evidence in US Army studies on bio-warfare that our sources say are in motion now.

https://ntp.niehs.nih.gov/ntp/ohat/sarin/sarin_draft20181219_508.pdf

The US has been relocating Global Hawk aircraft to Sicily, ostensibly to be used to spy on Russia through missions over the Black Sea.  However, other missions have been flown, Northern Italy, or across Northern Iran.  We know the US and Israel have developed the capability of dropping microdrones, with a self-destruct capability that can spread diseases like SARS, Swine Flu and the various strains of Bird Flu and crop diseases.  We also have evidence such devices have been used against China on 6 or more occasions.  We now have a new and far more sinister report.

We have a report from Russian intelligence that says the US is using low dose Sarin gas to mimic pandemic diseases.  They say they have evidence that in Iran and Italy sarin gas in extremely small concentrations has been in the food, water, and environment in minuscule concentrations.

These concentrations cause, over a two week period, the onset of pneumonia and a respiratory disorder.  Larger concentrations cause heart disease and mimic heart failure.

Starting in 1996 and then ramping up to an extreme level in 2006, the US-financed studies on various nerve agents and hallucinogens which could be dropped by drones, now by micro-drones such as Israel has developed.

Parallel studies were made on crop diseases but most included diseases that could move from livestock to humans, giving a ‘cover and deception’ cause that could be explained away through controlled press assets, which means ‘all of them.’ 

We will also add that we have found extensive CIA and Mossad/Shen Bet complicity in media manipulation.  Worse is the recent, March 19, 2020, piece by Mitch Prothero in Business Insider.  Prothero is a “go-to” guy for reliable propaganda from the CIA.

Mitch Prothero

and Business Insider regularly launders CIA promoted conspiracy theories to the “unwashed.”  It is, in fact, one of their most successful operations.  The article we refer to is an attempt to “close the barn door after the horse has run off” scheme involving the CIA’s bioweapons/Sarin lab in Tbilisi, Georgia that has been busted time and time again for leaking Swine Flu into the community near the facility and making Sarin gas to be used in Syria.

For years, Russia targeted conspiracy theories at a US-funded lab on the frontline of coronavirus testing

Here’s another one worse than the others, check the bio of the author, a food service worker.  The site was 2 years of short posts on prison reform, 50 words or so, then this showed up, which ended up on Trump’s desk as a manifesto for his blame China policy.  Ultra-curious:

Logistical and Technical Exploration into the Origins of the Wuhan Strain of Coronavirus (COVID-19)

An investigative team from VT/New Eastern Outlook and Russia24 not only got plans for the weapons facility but was eventually allowed in to film.  The result was devastating to the US bio-war capability in the region which has since recovered.

• Richard E. Lugar: Body of Evidence Suggests New US Biological Warfront Opening
• US-funded “Disinformation Oversight” of Bio Weapons Prevention Programmes in Georgia

We also know the CIA has repeatedly tested both chemical and biological weapons, often in New York City, on the subway, according to a New York Times article that covers the Senate hearings on this. (below/archival)

We also know the CIA tested low dose Sarin gas on the New York Subway in 2008, releasing gas at the Bowling Green station near midnight, along with other stations that were “end of the line.”  Some dropped immediately, others struggled up the steps and recovered in minutes while some suffered long-term illness (perhaps worse).

This program, documented by the Senate, was the precursor to the CV 19 augmentation exercises, Italy, Spain, Iran, and China.  Greece may well soon be targeted as well, based on geopolitical analysis and the pattern we have seen thus far.

We might also add that medical evidence from Italy and China shows far too many patients that don’t respond to treatments, far too many anecdotal deaths, then fall within baseline parameters for typical respiratory patients.

Someone will write a paper on this if not “accidented” first.

We begin:

Nerve Agents

The nerve agents are a group of, particularly toxic chemical warfare agents. They were developed just before and during World War II and are related chemically to the organophosphorus insecticides. The principal agents in this group are:

• • GA – tabun
  • GB – sarin
  • GD – soman
  • GF – cyclosarin
  • VX – methylphosphonothioic acid

The “G” agents tend to be non-persistent whereas the “V” agents are persistent. Some “G” agents may be thickened with various substances in order to increase their persistence, and therefore the total amount penetrating intact skin.

At room temperature, GB is a comparatively volatile liquid and therefore non-persistent. GD is also significantly volatile, as is GA though to a lesser extent. VX is a relatively non-volatile liquid and therefore persistent. It is regarded as presenting little vapor hazard to people exposed to it. In the pure state nerve agents are colorless and mobile liquids. In an impure state nerve agents may be encountered as yellowish to brown liquids. Some nerve agents have a faint fruity odor.

In 1996, a US Army study examined the question:

Are there observable long-term health effects associated with exposure to Sarin (GB) and mustard at concentrations below that needed to cause acute signs, symptoms, or injury?

Here are the findings of that study (emphasis added):

FINDINGS

SARIN (GB)

GB is a nerve agent and is chemically known as isopropyl methyl phosphonofluoridate. It is a colorless and odorless liquid when pure; the vapor is also colorless. GB evaporates at approximately the same rate as water. Like other nerve agents (soman, tabun, VX), GB is a highly toxic organophosphate that irreversibly binds to acetylcholinesterase. As a result, acetylcholine accumulates at neuromuscular junctions and causes a loss of function at these junctions. This interferes with the fundamental mechanism required for the normal function of the central nervous system and the peripheral nervous system, that is, the transmission of a nerve impulse. While the great majority of effects are due to the anticholinesterase actions of GB, not all effects are related to this characteristic.

Marrs, Maynard, and Sidell (1996) categorize the signs and symptoms of GB intoxication into three groups, muscarinic, nicotinic and central. The muscarinic signs and symptoms result from increased activity of the parasympathetic system and include miosis, dim vision, salivation, bradycardia, lacrimation, abdominal cramps, diarrhea and rhinorrhea. Nicotinic effects include pallor, tachycardia , hypertension, muscle fasciculation and weakness. Central nervous system effects include headache, anxiety, difficulty concentrating, restlessness, confusion, convulsions, and respiratory depression or paralysis, which can lead to death.

Signs and symptoms can be observed regardless of exposure route, but the intensity and sequence is influenced by the route of exposure. Skin exposure may cause localized sweating and fasciculations first. Vapor exposure where the eyes and respiratory tract may come into contact with GB first, may result in miosis, rhinorrhea, and tightness of chest first. Respiratory exposure appears to result in symptoms faster than skin exposure.

During vapor exposure studies and unintentional vapor exposures, the first signs and symptoms are usually miosis, rhinorrhea and/or chest tightness (Sidell, 1992). In fact, early studies often defined an individual as “exposed” when that person had at least one of these symptoms. Persons in the same area, without any health complaints, were not considered exposed or “hit.” This is the first of a multitude of methodologic problems related to the question at hand. Only those who had clinical signs or symptoms would be studied and documented. Anyone else, even if they were in the same area, would not be considered exposed and would not be examined.

The amount of GB necessary to cause initial clinical signs and symptoms is debatable but has been estimated to be approximately 2-3 mg-min/m3. This is known as the Ct, or the concentration of agent vapor in air as milligrams per cubic meter times the time of exposure in minutes (Sidell, 1992). McKee and Woolcott (1949) report that a single exposure to a Ct of 3.3 mg-min/m3 (for 40 minutes) is the minimum dosage necessary to produce effects in men. However, they also state that chemical analysis of the agent indicated that the concentration actually given was approximately 75% of the intended dose, therefore the Ct would actually be approximately 2.5 mg-min/m3. When the exposure time was reduced by 50% (20 minutes, Ct approximately 1.2 mg-min/m3), a single dose did not produce symptoms, but the same report indicated that exposure to a Ct of 0.6mg-min/m3 (1 minute) also caused detectable symptoms. It appears that a single exposure of man to a very small amount of GB will produce observable acute signs and/or symptoms. It is also important to note that exposure with a Ct derived over a longer period of time (e.g. 240 minutes vs 20 minutes) will cause fewer or less severe signs and symptoms since there is some detoxification that occurs during the long period of exposure.

As will be described below, there are essentially no controlled human studies in which men were exposed to doses calculated to avoid symptoms and where these men were followed over extended periods of time. Several studies utilizing doses that did cause acute symptoms, several unintentional high-level exposure investigations, and animal studies can be used to make general suggestions regarding the long-term health risks associated with low-level exposure to GB.

So, in 1996, there was no data on low-dose exposure to Sarin, therefore studies would need to be conducted to gather such data. The US Army came to the same conclusion (emphasis added):

Conclusions

Sarin, or GB is a highly toxic organophosphate nerve agent that can cause mild, reversible signs and symptoms at low doses, and death at doses that are not too much greater. There is no scientific information that directly answers the entire question: Are there observable long-term health effects associated with exposure to Sarin (GB) at concentrations below that needed to cause acute signs, symptoms, or injury? Extrapolation from a variety of sources, not designed to answer this particular question, was utilized. Some studies are clearly negative for a particular health effect at higher doses than that of concern to this question. These provide confidence that there is no increased specific health risk at the low doses under question.

GB does not have carcinogenic or mutagenic properties. While the teratology literature is less clear, it appears that GB is not a teratogen. Therefore, no increase in birth defects or cancer would be expected from low-dose, short-duration exposure to GB. A follow-up of a cohort of men exposed to GB found no significant increase in hospitalizations, reported health problems, mortality, or another measured endpoint.

There remains some question as to the validity of concerns regarding changes in EEG patterns long after GB exposure. Also, it is unclear what such changes really mean regarding the function of the soldier if those long-term EEG changes actually occur.

It is prudent to suggest that further research into the long-term effects of low dose GB exposure (including doses that do not result in acute signs or symptoms) on the EEG of primates be undertaken.

The two key sentences:

There is no scientific information that directly answers the entire question: Are there observable long-term health effects associated with exposure to Sarin (GB) at concentrations below that needed to cause acute signs, symptoms, or injury?

They don’t know what the effects of low-dosage sarin exposure are.

It is prudent to suggest that further research into the long-term effects of low dose GB exposure (including doses that do not result in acute signs or symptoms) on the EEG of primates be undertaken.

So they recommend carrying out studies.

The Army unit responsible for such studies is based at the Aberdeen Proving Ground in Maryland:

United States Army Medical Research Institute of Chemical Defense (USAMRICD) the nation’s leading science and technology laboratory in the area of medical chemical countermeasures research and development. With sophisticated laboratories located at Aberdeen Proving Ground, Maryland, USAMRICD manages a diversified portfolio of medical chemical warfare agent research projects for the Department of Defense and other Federal Agencies.

The USAMRICD did indeed carry out a study into low-dose Sarin exposure and published the results in 2006:  The effects of repeated low-dose sarin exposure

2006 Sep 1;215(2):119-34. Epub 2006 Mar 23.

The effects of repeated low-dose sarin exposure.

Shih TM*, Hulet SW, McDonough JH.
Author information

• • Pharmacology Branch, Research Division, U.S Army Medical Research Institute of Chemical Defense, Aberdeen Proving Ground, MD 21010-5400, USA. tsungming.a.shih@us.army.mil

Abstract

This project assessed the effects of repeated low-dose exposure of guinea pigs to the organophosphorus nerve agent sarin. Animals were injected once a day, 5 days per week (Monday-Friday), for 2 weeks with fractions (0.3x, 0.4x, 0.5x, or 0.6x) of the established LD(50) dose of sarin (42 microg/kg, s.c.). The animals were assessed for changes in body weight, red blood cell (RBC) acetylcholinesterase (AChE) levels, neurobehavioral reactions to a functional observational battery (FOB), cortical electroencephalographic (EEG) power spectrum, and intrinsic acetylcholine (ACh) neurotransmitter (NT) regulation over the 2 weeks of sarin exposure and for up to 12 days postinjection. No guinea pig receiving 0.3, 0.4 or 0.5 x LD(50) of sarin showed signs of cortical EEG seizures despite decreases in RBC AChE levels to as low as 10% of baseline, while seizures were evident in animals receiving 0.6 x LD(50) of sarin as early as the second day; subsequent injections led to incapacitation and death. Animals receiving 0.5 x LD(50) sarin showed obvious signs of cholinergic toxicity; overall, 2 of 13 animals receiving 0.5 x LD(50) sarin died before all 10 injections were given, and there was a significant increase in the angle of gait in the animals that lived. By the 10th day of injection, the animals receiving saline were significantly easier to remove from their cages and handle and significantly less responsive to an approaching pencil and touch on the rump in comparison with the first day of testing. In contrast, the animals receiving 0.4 x LD(50) sarin failed to show any significant reductions in their responses to an approaching pencil and a touch on the rump as compared with the first day. The 0.5 x LD(50) sarin animals also failed to show any significant changes to the approach and touch responses and did not adjust to handling or removal from the cage from the first day of injections to the last day of handling. Thus, the guinea pigs receiving the 0.4 and 0.5 x LD(50) doses of sarin failed to habituate to some aspects of neurobehavioral testing. Spectral analysis of EEG data suggested that repeated sarin exposure may disrupt normal sleeping patterns (i.e., lower frequency bandwidths). While these EEG changes returned to relative normalcy 6 days after the last injection in animals receiving 0.4 x LD(50) sarin, these changes were still observed in the animals that received 0.5 x LD(50) sarin. Ten to twelve days after the last sarin injection (in 0.4 x LD(50) group only), neurochemical data showed that striatal choline levels were reduced in comparison to the saline group. At this time, atropine sulfate (5 mg/kg, i.p.) challenge resulted in a transient elevation in striatal ACh levels in animals exposed to repeated 0.4 x LD(50) sarin as well as in control animals. No evidence of brain or heart pathology was found in any guinea pig that survived all 10 sarin injections.

PMID: 16556454
DOI: 10.1016/j.taap.2006.02.003

The sequence of symptoms varies with the route of exposure. While respiratory symptoms are generally the first to appear after inhalation of nerve agent vapor, gastrointestinal symptoms are usually the first after ingestion. Tightness in the chest is an early local symptom of respiratory exposure. This symptom progressively increases as the nerve agent is absorbed into the systemic circulation, whatever the route of exposure. Following comparable degrees of exposure, respiratory manifestations are most severe after inhalation, and gastrointestinal symptoms may be most severe after ingestion.

Naval Air Station Sigonella, just outside Catania on the Italian island of Sicily.

It is a 1,000km 2-hour flight to Milan for a Global Hawk drone flying from Sigonella

Northrop Grumman RQ-4 Block 40 Global Hawk, which entered service with the US Air Force in September 2013. This latest variant of the drone has synthetic-aperture radar and ground moving target indicators. Five of this unmanned craft are based at Sigonella.

New NATO surveillance drones bet on Italian safety ruling

The Five RQ-4D drones based at Sigonella became operational at the end of 2019 with the NATO AGS (Alliance Ground Surveillance).

WASHINGTON, Sept. 18 (2016)—Army scientists secretly spread simulated biological poison on two subway lines in Manhattan in the mid-nineteen-sixties to test the vulnerability of the New York subway system to a biological warfare attack, a Department of Defense engineer testified today.

Charles Senseney, a project engineer who developed weapons such as an electric poison dart gun and a system to spread biological poison from a fluorescent bulb, told the Senate Select Committee on Intelligence that he took part in the New York “vulnerability study” as one of many such efforts aimed at testing the dangers of biological warfare.

His account provided the first substantial details of the subway project, which was disclosed Tuesday in testimony and documents submitted to the Senate panel by William E. Colby, the Director of Central Intelligence.

Mr. Senseney said the studies. conducted by the staff of the Army laboratories at Fort Detrick, Md., had been performed on behalf of the Army and the Central Intelligence Agency. They included tests at the White House, the Pentagon, a Food and Drug Administration building in Washington, and McGuire Air Force Base in New Jersey.

In a meeting with reporters after today’s hearing, Mr. Senseney gave this account of the New York City subway experiments:

Possibly in 1966 or 1967, Mr. Senseney and 20 other Fort Detrick employees were sent to New York for the two‐week test. The “operator” of the test threw “bulbs” of a simulated biological poison on the tracks of two subway lines. He said he believed, but could not be sure, they were the Sixth and Eighth Avenue lines in Manhattan.

The bulbs burst and the wind of the passing subway trains spread the simulated poison along the tracks. He said that in the short time it took for two trains to pass the simulated poison was spread from 15th Street to 58th Street.

Mr. Senseney’s conclusion, which appeared to be the conclusion of the experiment, was that the subway system could “not be safeguarded against” this type of attack. If the attack was carried out during rush hours, he said, it would “put New York out of commission.”

Mr. Senseney said his role in the experiment was as a “sampler.” He and part of the team rode the subways with a sampling device t,o test the spread of the simulated poison. His was kept on his belt with the appearance of a photographic‐light meter, he said.

City Officials Not Told

He said the other detection devices had been hidden in pocketbooks and other camouflages so the subway passengers would not know what was being done. He said that to his knowledge — and Senate committee documents appear to confirm this —neither the New York City government nor the Transit Authority officials were aware that such a test was being conducted.

He said that the stimulant, which he declined to identify. was harmless. Several biologists, however, said it was impossible to tell if the simulant was entirely harmless without knowing what it was.

Mr. Senseney said that “depending on the agent” used, someone who wanted to attack the subways could either introduce material to kill large numbers of the passengers or make them ill. He said the tests were conducted between rush hours, but that a real attempt to disable the city would call for an attack during rush hours.

Mr. Senseney said the New York project was part of a broader effort to discover how vulnerable the United States was to germ warfare, stretching from the early nineteen‐sixties.

He knew of, but did not participate directly in, tests at the White House, Mr. Senseney said. He added that the White House experiment had revealed that the building had faulty air filters that made it extremely vulnerable to a biological attack,

Dye In Water System

Mr. Senseney confirmed under questioning by Senator Gary Hart, Democrat of Colorado, that the Fort Detrick labs had secretly placed a colored dye into the water system of a Washington building used by the Food and Drug Administration to test how fast the occupants could be killed or incapacitated by introducing a biological agent into the water system. Mr. Senseney said he had developed a special drill that allowed the dye to be punched into a water pipe without leakage or change in the water pressure.

Mr. Senseney said that his specialty at the Fort Detrick laboratories was making “hardware” devices to deliver poisons and biological agents.

He said he developed the “M‐1” dart gun system, an apparatus for firing poison darts. The electric dart gun shown by the C.I.A. at Tuesday’s hearing was one of the guns developed as a by‐product of this system, Mr. Senseney said.

He said that his unit, the Special Operations Division at Fort Detrick, had received assignments to crew e exotic weaponry. Most of the assignments came from the Army, mainly the Special Forces, he said, though he recalled one request from the C.I.A. to develop a hand‐held dart gun that could shoot a poison dart into a dog without leaving a trace.

Use in Asia Reported

Authoritative intelligence sources said that poison darts had been used operationally in Asia to “incapacitate” guard dogs at installations the C.I.A. wanted to enter surreptitiously. The poison did not kill the dogs, these sources said; instead, it put them out for several hours but left no trace so that examination would not reveal the dogs had been out of action.

Mr. Senseney said that the agency had asked for about 50 of these weapons and that he believed the agency had used them operationally. On several occasions, he said, he delivered the weapons to agency officials and they were returned six or seven weeks expended.

“We didn’t get any feedback [from the agency] so you didn’t know whether the device worked or not,” Mr. Senseney said.

Under questioning by Senator Walter Huddleston, Democrat of Kentucky, Mr. Senseney acknowledged that the system could have been used to kill human beings and he could not rule out the possibility that this had been done.

He said he had developed darts that would go through clothing and enter human beings without a trace. He said that one device had been developed for the Vietnam war hut had not been put into production quickly enough to be utilized.

Too ‘Exotic’ For Use

Mr. Serneney said he had also developed dart launchers that appeared to be walking canes and umbrellas, as well as a device that fitted into a fluorescent bulb and spread a biological poison when the light was switched on. Later, he told newsmen it could easily “take out” more than 100 newsmen and staff members as well as policemen, Senators and the public.

Mr. Senseney was called to testify in the committee’s continuing inquiry into why the C.I.A. failed to comply with the 1969 Presidential order to destroy biochemical weapons.

According to witnesses over the last three days, the agency discovered earlier this year about 11 grams of a lethal shellfish toxin in a storeroom at one of its Washington laboratories. This material should have a beer. destroyed under the conditions of a Presidential order framed in November 1969, and sent to Federal agencies in February 1970.

Dr. Nathan Gordon, the agency official in charge of the poison, testified that he decided to move the toxin from Fort Detrick to the agency lab in 1970 after receiving a call from Mr. Senseney offering the stockpile to him.

Mr. Senseney denied today that he ever made such a call. He said he did not know why the toxin was not destroyed but said he presumed it was because the agency wanted it retained.

Discrepancy Found

It was still unclear after today’s session why 11 grams was on hand in 1975 because the inventories made in 1970 indicated that the agency had a stockpile of only 5.9 grams.

Senator Richard S. Scheweicher, Republican of Pennsylvania, has suggested by his questioning that other Federal agencies may have surreptitiously avoided the Presidential order by sending their supplies of the toxin to the C.I.A.

The committee will continue the investigation, its chairman, Senator Frank Church, Democrat of Idaho, said.